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The rationale behind the administration of l-ARG as a suitable treatment for vasculogenic ED relies on the matter of fact that l-ARG is an important donor of NO, which is a prominent molecular mediator of the penile erectile process, for the ability to induce cavernous smooth muscle relaxation, and particularly to contribute to the cavernous arteries performance and cavernous blood flow, crucial for penis erection [4, 7]

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Anti-Mullerian hormone promotes pre-antral follicle growth, but inhibits antral follicle maturation and dominant follicle selection in primates